Stop Snoring and Obstructive Sleep Apnea

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So the relationship with sleep-disordered breathing, we have to consider snoring what is it, but the exchange of a large volume of air through a narrow space. Two things are going on here: the exchange of a large volume of air. If we breathe heavy during the day, we breathe heavy during our sleep and a second aspect is the size of the space, the diameter of the airway. We kind of consider one without your. Can you imagine a plumber coming to your house and the plumber has to determine the diameter of the pipe bringing water from se11 aspect of the house, one part of the house to the Iraq and you imagine the plumber just choosing the size of a pipe of Random, without considering the amount of water that’s expected to go through it, the two go hand-in-hand and we’re breathing. They also go hand-in-hand. Large breathing volume, narrowed space, two types of snoring snoring with them out open, very easy to address. All you have to do is get the mouth closed. The second type of snoring is high upper airway resistance, turbulent airflow and the names of Florence, the more difficult one. We show people how to decongest your nose. We show people have to bring their breathing volume down towards normal, no devices, no gadgets, just simple feedback. We look at an individual’s breathing and we give them simple, breathing exercises which would practice as a group and I’d really like you to practice them, because I can talk about the brush until you experience the effect you know it gives you a better perspective hours and Of course, the next progression is obstructive: sleep apnea normal minute volume. If you were to look, this is a 2009 book. If you were to look at normal volume, it’s four to six leaders of our permission. So generally, we have 12 racks, each practice, half liter and that’s giving us our four to six liters of air that is considered normal. But when we look at ask me and the reason that I’m bringing your password because I’m going to tie in a small obstructive sleep, apnea, there’s something going on with asthma, that’s also going on in osa, and we have to find out. Why wash is the relationship? But people would ask my breathing between 10 and 15 liters of our permission I was over pointed out earlier on. He says we live without food for weeks we live without water. For days we live without air for just a few minutes. You all know how much food you should be using everyday. You all have an idea of how much water you should be drinking, but how much air should you be breathing? Is it hefty to breed to to treat times the volume of air every minute every hour? Every day in a medical textbook, normal breathing is considered 426 leaders of our per minute. That’S here. If somebody walks into a doctor’s surgery and are having a panic, grass Metaxa can be having 20 to 30 liters of air per minute. That’S here, that’s recognized normal breathing is recognized. Excessive breathing is during a panic. Attack is recognized. Nobody is looking at the peace in the Middle if you get very sick and a short-term by breathing twenty to thirty leaders of our permission for my question is well. If you’re breathing to treat times more than what you should be every minute every hour every day, are you also going to get sick and it’s not just that asthma is causing over breathing it’s the feedback that over breathing is literally sucking, moisture out of the airways And causing inflammation to feed back into it as a feedback loop, and I also believe the same feedback loop is would always say, never studied very seldom minute volume obstructive sleep apnea. How much air does the individual breed in this paper? Here they looked at 20 obese man would always say a normal lung function and they were found to be breathing 15 liters of air per minute minute volume, 15 liters normal, is for 26 they’re breathing two to three times more, the next in the same paper 12.8. These people have overlap syndrome, Dave couple o SI and copd, but they’re still breathing excessively, and when we look I’d always say we have to consider flow, i’m just going to play a short video obstructive. Sleep apnea occurs when the upper Airways collapse, resulting in holding of the breath during sleep after a period of time, the sleeper partially wakes up gasping for air this cycle of breath, holding followed by gasping, can occur many times an hour during sleep in addressing obstructive sleep. Apnea two factors need to be considered. The first is airway size and the second is the amount of air that we habitually breathe in relation to airway sighs, the narrower the upper airway, the greater than negative pressure as air is drawn into the lungs habitual mouth breathing. During childhood can alter the shape of the face resulting in small or receiving jaws. This reduces airway, sighs and increases the risk of lifelong sleep apnea, as we get older loss of muscle tone in the upper airway also contributes to holding of the breath during sleep to help open the upper airway. It is important not to sleep on the back and also to breathe through the nose. The second consideration in obstructive sleep apnea is the amount of air that we breathe. It is very common for people to habitually breathe too much air, as indicated by audible breathing often through the mouth. Heavy breathing during sleep increases negative pressure in the upper Airways causing collapse. Imagine inhaling a large volume of air through a collapsible paper straw, the heavier one breeze, the greater the likelihood of the walls of the strong collapsing inwards. On the other hand, breathing light through the straw causes less negative pressure, thereby reducing the risk of collapsing. The walls breathing re-education to reset the breathing Center in the brain to a lighter breathing volume significantly reduces the occurrence of obstructive sleep. Apnea. Combining this with my own functional therapy to strengthen the upper Airways, enhances the benefits resulting in a significant reduction in apneas and hypopnea. Ok, so we’re going to be based on the Bernoulli principle and basically this states that this fluid flows and negative pressure develops at the periphery of the flow but, most importantly as flow lasting as flow velocity increases. So does the negative pressure so the best way to imagine it is breathing through a paper straw, collapsible paper straw, the upper Airways is collapsible. If you breathe hard you’re going to cause negative pressure on the inner walls of the Airways closing collapse, and the other aspect about this is that if positive pressure helps open up the airway, reducing negative pressure should also help ensure that the airway will stay more open And the second factor of this is the venturi effect, and this is looking at airway sighs and basically it’s a given volume of fluid moves through a conduit of decreasing diameter. That velocity of the fluid will increase the narrower the upper airway, the greater the velocity there’s. Two aspects going on: we have the Bernoulli principle, which is solely looking at our air volume, breathing volume and we have the venturi effect, which is looking at every size and when we look wash is obstructive. Sleep apnea, but collapse of the upper airway occurs. If the negative upper airway pressure generated by inspiring pump muscles exceeds the dilating force of these upper airway muscles and that definition is taken from a number of papers looking as obstructive sleep apnea again, let’s look at breathing. This here is, what’s called a starling resistor much model, and it’s very interesting in terms of. If there’s an obstruction in the front of the airway visa, be the nose, the nose is blocked and it was difficult to breathe through the nose. It will increase the negative pressure further on the airway, so we’re talking about breathing volume, we’re also talking about nasal obstruction, so it behaves like a starting resistance and obstruction at the inlet. The nasal airway produces collapsing forces that are manifest downstream and the collapsible segment deference. So the basic message so far as breathe through the nose and breathe lightly.

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